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T cell undergoes proliferation, differentiation and activation of the complete immune response. Mitogen-induced proliferation of T small dark by the mitogen, II A 1, IL-2 signal start of three mitogen-stimulated T cells into the G1 phase, M produce IL-1 acts on T cells to produce IL-2, membrane expression of IL-2 stimulated IL-2 receptor leaving cells into the s of, 1I-2 signaling role of the T cell junction ● If completed the cell cycle. There are several additional experiments show that V can be restricted to inhibit COX-lymphocytes and M + produced PGE. To increase M ÷ produce IL-1, T lymphocytes produce IL-2, stimulate lymphocyte proliferation. Some academics have also boost the immune response of V anti-oxidant challenge. Corwin and other immune stimulation seems independent of V antioxidant. Thus v mode of action is not simply the role of antioxidants. 4.3 The role of immune function in old age old age reduced immune function. Sakai and other reports, fed conventional feed Ageing proliferative response of rat spleen cells was significantly reduced, and presumably with the lymphocyte proliferation or the weakening of M function. It was also confirmed. aged mice remained activated lymphocytes AM macrophage activating factor production (MAF) capacity However, MAF aged spleen lymphocytes or in rat than the amount of low, so even old rats maintained the response to the MAF, but MAF does not produce enough volume to activate the M, can lead to significant reduction in cellular immune function. Mevdani other canthus found that elderly blood PGE! levels were significantly higher, and with the decreased cellular immune function in elderly people speculated that the elderly look askance so small roar proliferation may be due to the weakening of 111, reduce the level of M t to produce less stimulation of lymphocytes, but it may be due to the promotion of M generated PEG! the result. 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